Items where Author is "Bouzakri, K."

Article

Timper, K. and Dalmas, E. and Dror, E. and Rutti, S. and Thienel, C. and Sauter, N. S. and Bouzakri, K. and Bedat, B. and Pattou, F. and Kerr-Conte, J. and Boni-Schnetzler, M. and Donath, M. Y.. (2016) Glucose-Dependent Insulinotropic Peptide Stimulates Glucagon-Like Peptide 1 Production by Pancreatic Islets via Interleukin 6, Produced by α Cells. Gastroenterology, 151 (1). pp. 165-179.

Biason-Lauber, A. and Boni-Schnetzler, M. and Hubbard, B. P. and Bouzakri, K. and Brunner, A. and Cavelti-Weder, C. and Keller, C. and Meyer-Boni, M. and Meier, D. T. and Brorsson, C. and Timper, K. and Leibowitz, G. and Patrignani, A. and Bruggmann, R. and Boily, G. and Zulewski, H. and Geier, A. and Cermak, J. M. and Elliott, P. and Ellis, J. L. and Westphal, C. and Knobel, U. and Eloranta, J. J. and Kerr-Conte, J. and Pattou, F. and Konrad, D. and Matter, C. M. and Fontana, A. and Rogler, G. and Schlapbach, R. and Regairaz, C. and Carballido, J. M. and Glaser, B. and McBurney, M. W. and Pociot, F. and Sinclair, D. A. and Donath, M. Y.. (2013) Identification of a SIRT1 mutation in a family with type 1 diabetes. Cell metabolism, Vol. 17, H. 3. pp. 448-455.

Rutti, S. and Sauter, N. S. and Bouzakri, K. and Prazak, R. and Halban, P. A. and Donath, M. Y.. (2012) In vitro proliferation of adult human beta-cells. PLoS ONE, Vol. 7, H. 4 , e35801.

Bouzakri, K. and Plomgaard, P. and Berney, T. and Donath, M. Y. and Pedersen, B. K. and Halban, P. A.. (2011) Bimodal effect on pancreatic β-cells of secretory products from normal or insulin-resistant human skeletal muscle. Diabetes, Vol. 60, H. 4. pp. 1111-1121.

Ellingsgaard, H. and Hauselmann, I. and Schuler, B. and Habib, A. M. and Baggio, L. L. and Meier, D. T. and Eppler, E. and Bouzakri, K. and Wueest, S. and Muller, Y. D. and Hansen, A. M. and Reinecke, M. and Konrad, D. and Gassmann, M. and Reimann, F. and Halban, P. A. and Gromada, J. and Drucker, D. J. and Gribble, F. M. and Ehses, J. A. and Donath, M. Y.. (2011) Interleukin-6 enhances insulin secretion by increasing glucagon-like peptide-1 secretion from L cells and α-cells. Nature medicine, Vol. 17, H. 11. pp. 1481-1489.